And of course the Bill and Melinda Gates – funded GAVI text which is quite blatant, claiming 10% mortality, which I covered in my prior substack articleconcerning Monkeypox and fearporn.
I almost cannot believe that I am writing this, but since my original substack article on this topic, we had the reveal of an Event 201-style wargame exercisemodeled around a bioterror-related release of an engineered Monkeypox virus “caused by a terrorist attack using a pathogen engineered in a laboratory with inadequate biosafety and biosecurity provisions and weak oversight.” With amazing (coincidental?) prescience, the “table top exercise” of March 2021 (one year and three months into the Covidcrisis) models a Monkeypox bioterror attack initiated on May 15, 2022. Note the date of the CNN/Jake Tapper fearporn piece – May 20, 2022. The modeling deployed in the scenario upon which the “exercise” was based predicts 3.2 billion cases and 271 million deaths by December 01, 2023. Of course, the predictive accuracy of the simplistic public health models such as that used to support this scenario have repeatedly proven to be absolutely horrid, and these types of models should be either relegated to the trash heap (or ongoing dumpster fire) as unscientific speculation which is all too frequently weaponized by the fearporn peddlers such as CNN, MSNBC, NYT, Washington Post. By now we all know the usual USG and WEF-controlled media players.
As the Italian’s like to say:
As we now know, the amazing foresight of this modeled date immediately preceded a seminal WHO meeting which has just concluded, in which international health regulation (IHR) modifications which would grant the WHO unprecedented powers to bypass national constitutions (proposed on January 23, 2022 by the US HHS) were actively considered but tabled for a future meeting (~November 2022?) largely due to African nation concerns regarding infringement of national sovereignty. The stated purpose of the “exercise” was remarkably well aligned with the stated objectives and topics proposed by US HHS in the submitted IHR modifications:
In my prior substack entitled “Monkey Pox, Truth versus Fearporn”, I concluded the essay with the following caveat:
So, at the risk of oversimplification:
- Looks like the Monkeypox outbreak comes from a single original virus source. Following the teachings of the “Multiple working hypothesis” model for arriving at scientific “truth” (which was a core part of my education as a young scientist), a) this could be (for example) a “natural” single jump event from some infected animal into a single human somewhere in the world (who presumably had some relationship to the Maspalomas Gay Pride event). Or b) it could have come from an intentional release of a viral isolate. Mixed news – could be good or bad
- The authors have confirmed that this new outbreak virus maps to the “(less disease-causing) West African group (clade) of Monkeypox viruses. Good news
- This single source virus could have come from West Africa or could have come from United Kingdom, Israel or Singapore (consistent with either hypothesis a or b). Mixed news – could be good or bad
- Despite the sequences indicating that the virus is most closely related to those isolated in 2018-2019, it is significantly different. This could be due to natural evolution or due to laboratory engineering/gain of function “research” (consistent with hypotheses a) and b). Generally bad news. Basically, the authors are indicating that they believe that genome of this virus is either evolving more rapidly than one would expect from a double stranded DNA poxvirus, (left unsaid, or somebody has been messing around with it).
- The authors speculate that the pattern of mutations are consistent with the effects of a natural cellular protein with the abbreviated name of APOBEC3. For those who want to dive into the molecular virology of APOBEC3, here is a nice 2015 J Immunology review. For those seeking the “Cliff Notes” abridged version, see Wikipedia. For the obsessives or aficionados, note that APOBEC3 is associated with specific pattern of base changes- (C→ U). On the basis of their hypothesis regarding the potential role for APOBEC3, I infer that the authors must have detected a statistically significant fraction of C→ U changes in the current isolates relative to the 2018-2019 isolates. Mixed news – could be good or bad. Still does not differentiate between hypothesis a) or hypothesis b).
- Here is the rub. While APOBEC3 is associated with cellular resistance (yet another form of “innate immunity” – isn’t molecular virology and cell biology amazing!) to HIV (and presumably other retroviruses), a quick pubmed search reveals that Poxviruses are resistant to the mutational effects of APOBEC3! For example, see this 2006 paper published in “Virology”. Frankly, whether through lack of curiosity or fear of attack from government controlled media and journals, the failure of the authors to even mention this Virology article is a major oversight at best. My inference and interpretation? On the basis of this sequence analysis report from the INSA team cited above, to me this is looking more like a laboratory manipulated strain than a naturally evolved strain. Bad news.
- Furthermore, this double stranded DNA virus, infections by which have historically been self-limiting, appears to be evolving (during the last few days!) to a form that is more readily transmitted from human to human. Bad news.
In conclusion, the preponderance of current evidence is pointing towards a hypothesis for the origin of this outbreak which is increasingly consistent with prior “war game” scenario planning, remarkably akin to that which occurred during Event 201, which posits emergence of an engineered Monkeypox virus into the human population during mid-May of 2022.
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