Coronavirus deaths in the country have nearly reached a level where the virus will cease to qualify as an epidemic under Centers for Disease Control and Prevention rules, the federal agency reported on Friday.
The CDC qualifies a disease outbreak as an “epidemic” if the number of deaths attributable to the disease exceeds a certain percentage of total deaths per week. That threshold for pneumonia, influenza and COVID-19 fluctuates slightly depending on the time of year, ranging from around 7% at the height of flu season to around 5% during less virulent months.
CDC data indicate that deaths from those ailments began skyrocketing in the country around the second week of March, hitting a peak around early May and then plummeting quickly after that.
The latest data show that the percentage of deaths in the country attributable to those factors had as of the last week in June reached its lowest point since the end of last year, becoming “equal to the [current] epidemic threshold of 5.9%,” the CDC said.
The welcome news comes as fear over a “second wave” of the virus has gripped the U.S., with some states experiencing fresh surges of COVID-19 along with increased hospitalizations.
Though infections are significantly up in some places, deaths throughout the country have remained flat, due likely to several factors including a younger cohort of infections as well as improved treatment methods.
The original article was published in the Swiss magazine Weltwoche (World Week) on June 10th. The author, Beda M Stadler is the former director of the Institute for Immunology at the University of Bern, a biologist and professor emeritus. Stadler is an important medical professional in Switzerland, he also likes to use provoking language, which should not deter you from the extremely important points he makes.
The coronavirus is slowly retreating. What actually happened in the past few weeks? The experts have missed basic connections. The immune response against the virus is much stronger than we thought.
This is not an accusation, but a ruthless taking stock [of the current situation]. I could slap myself, because I looked at Sars-CoV2- way too long with panic. I am also somewhat annoyed with many of my immunology colleagues who so far have left the discussion about Covid-19 to virologist and epidemiologist. I feel it is time to criticise some of the main and completely wrong public statements about this virus.
Firstly, it was wrong to claim that this virus was novel. Secondly, It was even more wrong to claim that the population would not already have some immunity against this virus. Thirdly, it was the crowning of stupidity to claim that someone could have Covid-19 without any symptoms at all or even to pass the disease along without showing any symptoms whatsoever.
1. A new virus?
At the end of 2019 a coronavirus, which was considered novel, was detected in China. When the gene sequence, i.e. the blueprint of this virus, was identified and was given a similar name to the 2002 identified Sars, i.e. Sars-CoV-2, we should have already asked ourselves then how far [this virus] is related to other coronaviri, which can make human beings sick. But no, instead we discussed from which animal as part of a Chinese menu the virus might have sprung. In the meantime, however, many more people believe the Chinese were so stupid as to release this virus upon themselves in their own country. Now that we’re talking about developing a vaccine against the virus, we suddenly see studies which show that this so-called novel virus is very strongly related to Sars-1 as well as other beta-coronaviri which make us suffer every year in the form of a colds. Apart from the pure homologies in the sequence between the various coronaviri which can make people sick, [scientists] currently work on identifying a number of areas on the virus in the same way as human immune cells identify them. This is no longer about the genetic relationship, but about how our immune system sees this virus, i.e. which parts of other coronaviri could potentially be used in a vaccine.
So: Sars-Cov-2 isn’t all that new, but merely a seasonal cold virus that mutated and disappears in summer, as all cold viri do — which is what we’re observing globally right now. Flu viri mutate significantly more, by the way, and nobody would ever claim that a new flu virus strain was completely novel. Many veterinary doctors where therefore annoyed by this claim of novelty, as they have been vaccinating cats, dogs, pigs, and cows for years against coronaviri.
2. The fairy tale of no immunity
From the World Health Organisation (WHO) to every Facebook-virologist, everyone claimed this virus was particularly dangerous, because there was no immunity against it, because it was a novel virus. Even Anthony Fauci, the most important advisor to the Trump administration noted at the beginning at every public appearance that the danger of the virus lay in the fact that there was no immunity against it. Tony and I often sat next to each other at immunology seminars at the National Institute of Health in Bethesda in the US, because we worked in related fields back then. So for a while I was pretty uncritical of his statements, since he was a respectable colleague of mine.
The penny dropped only when I realised that the first commercially available antibody test [for Sars-CoV-2] was put together from an old antibody test that was meant to detect Sars-1. This kind of test evaluates if there are antibodies in someone’s blood and if they came about through an early fight against the virus. [Scientists] even extracted antibodies from a Lama that would detect Sars-1, Sars-CoV-2, and even the Mers virus. It also became known that Sars-CoV-2 had a less significant impact in areas in China where Sars-1 had previously raged. This is clear evidence urgently suggesting that our immune system considers Sars-1 and Sars-Cov-2 at least partially identical and that one virus could probably protect us from the other
That’s when I realised that the entire world simply claimed that there was no immunity, but in reality, nobody had a test ready to prove such a statement. That wasn’t science, but pure speculation based on a gut feeling that was then parroted by everyone. To this day there isn’t a single antibody test that can describe all possible immunological situations, such as: if someone is immune, since when, what the neutralising antibodies are targeting and how many structures exist on other coronaviri that can equally lead to immunity.
In mid-April work was published by the group of Andreas Thiel at the Charité Berlin. A paper with 30 authors, amongst them the virologist Christian Drosten. It showed that in 34 % of people in Berlin who had never been in contact with the Sars-CoV-2 virus showed nonetheless T-cell immunity against it (T-cell immunity is a different kind of immune reaction, see below). This means that our T-cells, i.e. white blood cells, detect common structures appearing on Sars-CoV-2 and regular cold viri and therefore combat both of them.
3. The failure of modellers
Epidemiologist also fell for the myth that there was no immunity in the population. They also didn’t want to believe that coronaviri were seasonal cold viri that would disappear in summer. Otherwise their curve models would have looked differently. When the initial worst case scenarios didn’t come true anywhere, some now still cling to models predicting a second wave. Let’s leave them their hopes — I’ve never seen a scientific branch that manoeuvred itself so much into the offside. I have also not yet understood why epidemiologists were so much more interested in the number of deaths, rather than in the numbers that could be saved.
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